Hemodynamic effects of isoproterenol and norepinephrine in acute cardiac tamponade

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Hemodynamic effects of isoproterenol and norepinephrine in acute cardiac tamponade.

The hemodynamic effects of isoproterenol infusion, 0.5 mug/kg per min were evaluated in eight intact anesthetized dogs during cardiac tamponade. During tamponade, the mean of pericardial pressures was increased from - 1.5 to 12.5 mm Hg, and the mean of right atrial pressures was increased from 1 to 12.4 mm Hg. Mean cardiac output fell from 144.8 to 44.8 ml/kg per min (P < 0.001), and rose to 10...

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Hemodynamic effects of nitroprusside and hydralazine in experimental cardiac tamponade.

Cardiac tamponade is associated with decreased cardiac output and increased systemic vascular resistance. Thus, vasodilator drugs might lower systemic resistance and increase cardiac output. Three groups of dogs were studied during tamponade. Group I received nitroprusside only; group II received blood transfusion and then nitroprusside; group III received hydralazine. In group I, nitroprusside...

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Hemodynamic effects of volume expansion in patients with cardiac tamponade.

BACKGROUND Volume expansion has been proposed as an alternative treatment for cardiac tamponade; however, the scientific evidence for this recommendation is very poor. METHODS AND RESULTS Forty-nine unselected patients (23 males; age 55+/-16 years) with large pericardial effusion and hemodynamic tamponade underwent fluid overload with intravenous administration of 500 mL of normal saline over...

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Cardiac tamponade: hemodynamic observations in man.

Hemodynamic studies were performed before and after pericardiocentesis in 19 patients with pericardial effusion. Right atrial pressure decreases significantly, from 16 +/- 4 mm Hg (mean +/- SD) to 7 +/- 5 mm Hg in 14 patients with cardiac tamponade. This change was accompanied by significant increases in cardiac output (3.87 +/- 1.77 to 7 +/- 2.2 l/min) and inspiratory systemic arterial pulse p...

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Cardiac tamponade in acute pancreatitis.

A 37-year-old woman was admitted to hospital with severe epigastric pain. She gave a four-month history of intermittent epigastric discomfort aggravated by fats. This had proved unresponsive to cimetidine, carbenoxolone, and diazepam. She did not drink alcohol. She was distressed, mildly jaundiced, and had marked abdominal tenderness. Her blood pressure was 130/70 mmHg and her heart rate 80/min...

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ژورنال

عنوان ژورنال: Journal of Clinical Investigation

سال: 1969

ISSN: 0021-9738

DOI: 10.1172/jci106007